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PLoS pathogens
Activating PKC-ε induces HIV expression with improved tolerability.
BiP/GRP78 is a pro-viral factor for diverse dsDNA viruses that promotes the survival and proliferation of cells upon KSHV infection.
Differences in phenotype between long-lived memory B cells against Plasmodium falciparum merozoite antigens and variant surface antigens.
Genomic epidemiology demonstrates spatially clustered, local transmission of Plasmodium falciparum in forest-going populations in southern Lao PDR.
A cohort-based study of host gene expression: tumor suppressor and innate immune/inflammatory pathways associated with the HIV reservoir size.
Infectious viral shedding of SARS-CoV-2 Delta following vaccination: A longitudinal cohort study.
Cbp1, a fungal virulence factor under positive selection, forms an effector complex that drives macrophage lysis.
Bartonella type IV secretion effector BepC induces stress fiber formation through activation of GEF-H1.
Relationship between CD4 T cell turnover, cellular differentiation and HIV persistence during ART.
Identification of unrecognized host factors promoting HIV-1 latency.